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Part 1 book “A concise textbook of oral and maxillofacial surgery” has contents: Introduction to oral and maxillofacial surgery, diagnosis in oral and maxillofacial surgery, management of medically compromised patient and medical emergencies, sterilization and infection control, armamentarium and their usage in oral and maxillofacial surgery,… and other contents.
http://dentalebooks.com A Concise Textbook of Oral and Maxillofacial Surgery http://dentalebooks.com http://dentalebooks.com A Concise Textbook of Oral and Maxillofacial Surgery Author Sumit Sanghai BDS (RGUHS) Lecturer Dr BR Ambedkar Institute of Dental Sciences and Hospital Patna, India Co-Author Parama Chatterjee BDS (RGUHS) ® JAYPEE BROTHERS MEDICAL PUBLISHERS (P) LTD New Delhi • Ahmedabad • Bengaluru • Chennai • Hyderabad • Kochi Kolkata • Lucknow • Mumbai • Nagpur • St Louis (USA) http://dentalebooks.com Published by Jitendar P V j Jaypee Brothers Medical Publishers (P) Ltd Corporate Office 4838/24, Ansari Road, Daryaganj, New Delhi 110 002, India, Phone: +91-11-43574357 Registered Office B-3, EMCA House, 23/23B Ansari Road, Daryaganj, New Delhi 110 002, India Phones: +91-11-23272143, +91-11-23272703, +91-11-23282021, +91-11-23245672 Rel: +91-11-32558559 Fax: +91-11-23276490, +91-11-23245683 e-mail: jaypee@jaypeebrothers.com, Website: www.jaypeebrothers.com Branches • 2/B, Akruti 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Square Kolkata 700 013, Phones: +91-33-22651926, +91-33-22276404, +91-33-22276415, Rel: +91-33-32901926 Fax: +91-33-22656075, e-mail: kolkata@jaypeebrothers.com • Lekhraj Market III, B-2, Sector-4, Faizabad Road, Indira Nagar Lucknow 226 016, Phones: +91-522-3040553, +91-522-3040554, e-mail: lucknow@jaypeebrothers.com • 106 Amit Industrial Estate, 61 Dr SS Rao Road, Near MGM Hospital, Parel Mumbai 400012, Phones: +91-22-24124863, +91-22-24104532, Rel: +91-22-32926896 Fax: +91-22-24160828, e-mail: mumbai@jaypeebrothers.com • “KAMALPUSHPA” 38, Reshimbag, Opp Mohota Science College, Umred Road Nagpur 440 009 (MS), Phone: Rel: +91-712-3245220, Fax: +91-712-2704275, e-mail: nagpur@jaypeebrothers.com USA Office 1745, Pheasant Run Drive, Maryland Heights (Missouri), MO 63043, USA, Ph: 001-636-6279734 e-mail: jaypee@jaypeebrothers.com, anjulav@jaypeebrothers.com A Concise Textbook of Oral and Maxillofacial Surgery © 2009, Sumit Sanghai, Parama Chatterjee All rights reserved No part of this publication should be reproduced, stored in a retrieval system, or transmitted in any form or by any means: electronic, mechanical, photocopying, recording, or otherwise, without the prior written permission of the authors and the publisher This book has been published in good faith that the material provided by authors is original Every effort is made to ensure accuracy of material, but the publisher, printer and authors will not be held responsible for any inadvertent error(s) In case of any dispute, all legal matters are to be settled under Delhi jurisdiction only First Edition: 2009 ISBN 978-81-8448-505-9 Typeset at Printed at JPBMP typesetting unit Rajkamal http://dentalebooks.com Dedicated to My father Mr Satyanarayan Sanghai and my mother Mrs Sassi Sanghai, for their continuous encouragement, understanding and support http://dentalebooks.com http://dentalebooks.com Foreword It is with great pleasure that I write this foreword for Dr Sumit Sanghai, an undergraduate student of mine who has done a commendable job of writing this book A comprehensive coverage of the subject based on the syllabus of DCI along with a lucid representation makes it a valuable aid to BDS students in the subject of Oral and Maxillofacial Surgery It is a concise compilation with self explanatory diagrams and well laid out tables He has explained the subject in simple sentence structuring making it easier to comprehend the concepts, facts and procedures The attractive outlay and organized presentation makes easy reading I wish him all the best, “God Bless” Ramdas Balakrishna BDS, MDS Oral and Maxillofacial Surgeon and Implantologist, Prof–Oxford Dental College and Hospital, Bangalore http://dentalebooks.com Foreword The efforts that have gone into the compilation of this text is commendable I congradulate these two young doctor, Dr Sumit Sanghai and Dr Parama Chatterjee for being a source of inspiration to numerous impressionable minds Deepika Kenkere BDS, MDS, FICOI, MAOMSI, MIAO Oral and Maxillofacial Surgeon and Implantologist Prof and Head-Department of Oral and Maxillofacial Surgery Sri Rajiv Gandhi College of Dental Sciences and Hospital, Bangalore http://dentalebooks.com FOREWORD I wholeheartedly congratulate Dr Sumit Sanghai on his endeavour to bring out this edition of “Concise Textbook of Oral and Maxillofacial Surgery” Oral surgical procedures have been ingrained deeply into every sophisticated dental practice This text fulfils the need for a concise and comprehensive book for the dental graduates The uniqueness of this book lies in the sequential manner in which the chapters have been dealt with I am sure that this edition will prove to be a valuable source of information for all dental graduates Arun Jacob Silas BDS, MDS Principal Prof and Head-Department of Pediatric Dentistry http://dentalebooks.com 122 A Concise Textbook of Oral and Maxillofacial Surgery Severe Infection • Trival + signs of toxicity – Paleness – Rapid respiration – Rapid thrombing pulse – Shivering – Fever – Lethargy – Diaphoresis (severe sweating) Fig 9.2: Spread of infection from infected mandibular third molar PATHWAY OF DENTAL INFECTION Extreme Infection • Trivial + sign of toxicity + CNS changes – Impaired eye movement/vision – Decreased level of consciousness • Meningeal irritation (severe headache, stiff neck, vomiting) • Edema of eyelids • Airway compromise • Difficulty in swallowing Diagnosis Lab Studies ODONTOGENIC INFECTION (GENERAL STUDY) Clinical Features (Signs and Symptoms) Mild Infection • Trivial → Inflammatory sign – Dolor – Calor – Rubor – Tumor – Loss of function • Lymphadenopathy • Pyrexia (fever) • Uncomplicated abscess: No laboratory studies are required • Complicated abscess: – The CBC count may reveal leukocytosis with neutrophil predominance – Obtain a blood culture (aerobic and anaerobic) before initiating parental antibiotic – Needle aspiration is indicated for gram stain and culture Imaging Studies • Uncomplicated abscess: No imaging studies are required • Complicated abscess: – Plain radiography represents the first level of investigation because it is readily available http://dentalebooks.com Infection of the Orofacial Region – Lateral and anteroposterior neck views may reveal a soft tissue neck mass that reveals abscess – Panoramic radiograph (pantomography) is helpful to indicate whether bone or teeth is involved – C.T scan with intravenous contrast is the most accurate method to determine the local relationship of the inflammatory process to the surrounding vital structures PROCEDURES Aspiration • Confirm presence of the abscess via needle aspiration • If pus is obtained, not aspirate more than or drops Leave the abscess as later, area is easier to find for further management • If pus cannot be aspirated, manage medically until a more localized infection develops • Incision and drainage may be performed only if pus can be aspirated • Packing a periapical abscess is generally not necessary Differential Diagnosis Three stages in progression of acute odontogenic infection: Periapical Osteitis or Apical periodontitis infection confined within alveolar bone Cellulitis - Infection spreads through bone, periosteum into soft tissue - No suppuration Stage of Abscess formation - Suppuration and localization within 72 hours of cellulitis • • • • 123 secure the airway via endotracheal intubation or tracheostomy Properly collect specimen for gram stain and culture Administer empiric antibiotic therapy Administer analgesia Hydrate the patient and use of treatment agent ( Hot packs are contraindicated in case of cellulitis as can lead to abscess) Surgical Care • The primary therapeutic modality is surgical drainage of any pus collection Incision and rupture of the abscess quickly accelerates resolution of the infection Abscess should be drained even if patient is toxic, since toxemia usually results from the absorption of degenerated tissue products and bacterial toxins • Emergent surgery is indicated in the operating room if the airway is threatened or if the pulse is deteriorating Consultations • Consult a dentist if the patient has an uncomplicated abscess • Consult a maxillofacial oral surgeon if the patient has a complicated abscess Diet Diet is as tolerated However, a soft bland diet is usually preferred Activity Activity is as tolerated Treatment TREATMENT OF ODONTOGENIC INFECTION Medical Care Treatment of odontogenic infection depends on: a Stage of infection b Physiologic response of the patient • Assess the airway upon respiratory distress, oropharyngeal tissue swelling or inability to http://dentalebooks.com 124 A Concise Textbook of Oral and Maxillofacial Surgery Sl No Stage of infection Periapical Osteitis a Infection confined within the bone b Tooth extruded from socket c Tooth painful to biting or percussions Response to infection Suggested treatment Non- toxic Prophylactic antibiotic and extraction of tooth Open pulp chamber and give supportive care (antibiotic, analgesic, hot application until patient is no longer toxic) Toxic (elevated temperature > 38° C, Chills, swelling Malaise, anorexia, W.B.C etc) Cellulitis a Swelling not sharply demarcated Non-toxic b Tissue have doughly consistency Toxic c No fluctuation Abscess a Swelling distinctly outlined Non-toxic b Tooth have firm, consistency Toxic c Prophylactic antibiotic and extraction of the involved tooth Open pulp chamber and give supportive care until patient is non-toxic Evaluation of pus by incision drainage and/or extraction of involved tooth Prophylactic antibiotic and drainage by incision and drainage and/or extraction Fluctuation elicited Note: If tooth to be conserve by RCT, pulp chamber should be opened PRINCIPLES FOR THE USE OF PROPHYLACTIC ANTIBIOTICS The operative procedure must have a risk of significant bacterial contamination and a high incidence of infection The organism most likely to cause the infection must be known The antibiotic susceptibility of the causative organism must be known To be effective and to minimize adverse effects, the antibiotic must be in the tissue at the time of contamination (operation), and it must be continued for no more than four hours after cessation of contamination The drug must be given in dosages sufficient to reach four times the MIC of the causative organism (MIC = Minimum inhibitory concentration of an antibiotic for a specific bacteria) PRINCIPLES FOR THE USE OF THERAPEUTIC ANTIBIOTICS Identification of the causative organism Determination of the antibiotic sensitivity Use of specific, narrow- spectrum antibiotics Use of least toxic antibiotic Patient drug history Use of bacteriocidal rather than bacteriostatic drugs Use of antibiotics with a proven history of success Cost of antibiotics Common Antibiotic Regime Used • Amoxycillin gm, hr prior to procedure + Amoxycillin 1.5 gm, hr after initial dose http://dentalebooks.com Infection of the Orofacial Region • For children, Amoxycillin 15 mg/kg, hr before procedure + Amoxycillin half initial dose, hr after initial dose • In pencillin sensitive patients, Erythromycin or clindamycin • In case of parenteral usage, Ampicillin, clindamycin, gentamycin or vancomycin PRINCIPLES OF INCISION AND DRAINAGE Knowledge of local anatomy of the area to be incised Incision should be made in the most dependent area (Incision should not be made in centre as causes necrosis and scarring) Wide incisions for drainage (has technical and esthetic problems) Incision placed in esthetically accepted area – Parallel to the skin folds or shadow of mandible “Hilton's method of abscess drainage” Closed blades of sinus forceps are thrust through the incision and into the abscess, ensures that no blood vessel or nerve is damaged Causes For The Failure in Treatment of Infection • • • • Inadequate surgical treatment Depressed host defenses Presence of foreign body Antibiotic problems: – Drug not reaching infection – Dose not adequate – Wrong bacterial diagnosis – Wrong antibiotic 125 INFECTION OF THE PULPAL AND PERIAPICAL TISSUES Etiology Physical A mechanical i accidental ii iatrogenic iii pathological iv aerodontalgia B Thermal i heat ii friction iii cold Chemical Bacterial i caries ii plaque iii anachoretic pulpitis STUDY OF SOME PULPAL AND PERIAPICAL INFECTIONS Focal Reversible Pulpitis or Pulpal Hyperemia It is an early, mild, transient pulpitis localized chiefly to the pulpal ends of the irritated dental tubules Features • Tooth is sensitive to thermal changes specially cold • Hyperactive to electrical changes • Clinically visible deep caries or large metallic restoration • Pain can be elicited which subsides after a transient period Chronic Hyperplastic Pulpitis or Pulp Polyp It is an excessive exuberant proliferation of chronically inflamed dental pulpal tissues http://dentalebooks.com 126 A Concise Textbook of Oral and Maxillofacial Surgery Features • Exclusively seen in children and young adults • Clinically visible large open carious lesion, pinkish red globular tissue seen protruding from pulp chamber • Generally insensitive to touch but may bleed on manipulation • Should be carefully differentiated from gingival proliferation Acute Pulpitis It is an extensive reversible or irreversible acute infection of the dental pulp Features History Pain Reversible pulpitis Irreversible pulpitis Slight sensitivity or occasional pain Momentary and immediate, sharp in nature, dissipates after removal of stimulus Localized No difference Constant or intermittent pain Continuous delayed onset, throbbing persists for minutes to hours after removal of stimulus Not localized Pain increases Location Change of posture Thermal test Responds immediately Electric pulp Early response test Percussion Negative Radiograph Negative Delayed response Early, delayed or mixed response Negative in early stages, later positive when periapex involve May show widening of PDL spaces Apical Periodontal Cyst or Periapical Cyst (Radicular Cyst, Bay Cyst) It is a sequence of periapical granuloma caused due to necrosis of pulpal tissues A true cyst is a pathological cavity lined by epithelial or nonepithelial tissues and is often filled with fluid Features • Generally asymptomatic but may show mild pain or sensitivity on percussion • Extraoral and/or intraoral swelling with or without expansion of cortical plates may be there • May lead to abscess, cellulites or even fistula Chronic Apical Periodontitis or Periapical Granuloma It is a localized mass of chronic granulation tissues associated with the root of an infected tooth or teeth Features • Sensitivity with mild or severe pain is felt which can be localized on percussion or while chewing • Extrusion of tooth from socket may be there • Inflammation, edema and swelling can be seen intra and extraorally Periapical and Periodontal Abscess Features Features Periapical abscess Periodontal abscess Cause Pain Swelling Pulpal infection Severe and throbbing In mucobuccal region, usually near apex of involved tooth Present Periodontal infection Severe and throbbing In attached gingiva Tenderness to percussion Sinus May be present discharge Mobility Seen at later stages Present May be present Seen even at earlier stages Pocket Single and narrow Multiple, wide coronally Pulp Necrotic and infected Vital Radiographic Localized bone loss Generalised bone loss feature which is mostly which is mostly horizontal and more vertical and more near the apex of near the coronal tooth portion of the tooth Treatment Root canal therapy Periodontal or extraction Intervention http://dentalebooks.com Infection of the Orofacial Region SPACES INVOLVED IN ODONTOGENIC INFECTIONS 127 iii Sublingual space iv Buccal space Secondary Spaces a Masseteric space b Pterygomandibular space c Superficial and deep temporal space d Lateral pharyngeal space e Retropharyngeal space f Prevertebral spaces g Parotid space Primary Spaces A MAXILLARY i Canine space ii Buccal space iii Infratemporal space B MANDIBULAR i Submental space ii Submandibular space PRIMARY MAXILLARY SPACES Spaces Canine fossa/ Infraorbital space Buccal Space Infratemporal Space i Location/Boundaries • Superiorly Infraorbital ridge Zygomatic Arch Zygomatic arch and infratemporal surface of greater wing of sphenoid • Inferiorly Canninus muscle Lower border of mandible Lateral pterygoid muscle • Anteriorly Orbicularis oris Orbicularis Oris, Zygomaticus Infratemporal surface of maxilla major, muscle over Ramus • Posteriorly Buccinator muscle Masseter muscle over ramus • Medially Anterolateral surface of Buccinator muscle and Medial pterygoid plate, lateral maxilla masseter muscle overlying pterygoid muscle, medial anterior border of ramus pterygoid muscle, lower part of temporal fossa and lateral wall of pharynx • Laterally - Skin, subcutaneous tissue and Ramus of mandible and temporalis muscle platysma ii Contents Buccal pad of fat, stenson's Medial pterygoid muscle, duct, facial artery lateral pterygoid muscle, pterygoid plexus of vein, maxillary artery, mandible nerve, middle meningeal artery iii Features Infection spreads from Infection spread from Trismus maxillary cuspid and from maxillary and mandibular premolars and molars nasal infection Parotid gland Swelling of cheek and upper Gum boil or prominent E/O Swelling of area and difficult swelling from infraorbital to open eye lip region till lower border of Obliteration of nasolabial fold, mandible drooping of angle of mouth, odema of lower eyelid Contd http://dentalebooks.com 128 A Concise Textbook of Oral and Maxillofacial Surgery Contd Spaces Canine fossa/ Infraorbital space Buccal Space Infratemporal Space iv Surgical treatment (incision and drainage) Approached through the mucosa of buccal vestibule in region of lateral incisor and canine Approached through cheek mucosa by horizontal incision near premolar and molar region Approached I/O from buccal vestibule opposite to 2nd and 3rd molar Approached E/O at the upper and posterior edge of temproalis muscle within the hairline Fig 9.3: Spreed infections to buccal space Fig 9.4: Cannine space and its boundries PRIMARY MANDIBULAR SPACES Spaces Sublingual space Submandibular space i Location/boundaries Superiorly Mucosa or oral cavity Inferiorly Mylohyoid muscle Medial aspect of mandible Mylohyoid muscle and the attachment of mylohyoid muscle Anterior and posterior bellies Suprahyoid portion of deep cervical fascia of digastric Anteriorly Lingual aspect of mandible Anterior belly of digasrtic and mylohyoid muscle Posteriorly At the midline, by body of hyoid bone Geniohyoid, genioglossus and styloglossus muscle Floor is formed by hyoglossus muscle Mylohyoid, hyoglossus and styloglossus muscle Medially Submental space Contd http://dentalebooks.com Infection of the Orofacial Region 129 Contd Spaces Sublingual space Submandibular space Laterally Lingual aspect of mandible Skin, superficial fascia, Lower body of mandible of platysma and superficial layer mandible and belly of of deep cervical fascia digastric ii Contents Geniohyoid, genioglossus, hyoglossus muscle Also contain submandibillar salivary gland and duct, sublingual salivary gland, lingual nerve and hyoglossal nerve Superficial lobe of Submental lymph nodes and submandibular salivary gland, anterior jugular veins submandibular lymph node, facial artery and vein iii Features – Infection spreads from mandibular anteriors, premolars and 1st molar – Extra orally, No swelling – Intra oral, painful firm swelling in floor of mouth – Pain, discomfort in deglutition, airway obstruction and affected speech Infection spreads from mandibular molars or secondary from sublingual space Firm, extraoral swelling below inferior border of mandible Tenderness, dysphagia, trismus and sensitivity of teeth to percussion Infection spreads from mandibular anterior teeth Distinct firm extraoral swelling along the midline beneath the chin Mobility and tenderness on percussion of teeth iv Treatment (incision and – Bilateral intraoral incision is made through the mucosa, drainage) parallel to wharton's duct at the base of alveolar process Bilateral through and through incisions made along the shadow of mandible through the skin, superficial fascia blunt dissection through platysma A blunt dissection is carried out by making a transverse incision in skin below the symphysis of mandible Fig 9.5: Spread of infection to submandibular space Submental space Fig 9.6: Spread of infection to sublingual space http://dentalebooks.com 130 A Concise Textbook of Oral and Maxillofacial Surgery Fig 9.7: Spread of infection of submental space MASTIGATORY SPACES Spaces i Sub-masseteric space Pterygomandibular space Temporal space (superfifcial and deep) Location/Boundaries Lateral pterygoid muscle Superficial temporal pouch in between temporal fascia and temporalis muscle • Superiorly Zygomatic Arch • Inferiorly Masseteric muscle attachment on the mandible • Anteriorly Anterior border of massetric muscle and Buccinator and fascia of parotid gland Pterygomandibular Raphe Deep temporal pouch lies between temporal muscle and skull • Posteriorly Parotid gland and posterior aspect of masseter Parotid gland —— • Medially Lateral aspect of Ramus of mandible Lateral aspect of medial pterygoid muscle —— • Laterally Medial border of masseter muscle Medial surface of Ramus —— ii Contents Muscles of mastication (massetric, lateral and medial pterygoid and temporalis) Communicates directly with Lingual nerve mandibular nerve, Inferior alveolar nerve infratemporal and pterygopalatine fossa Ramus of mandible Mandibular artery Buccal nerve, inferior alveolar nerve Mylohyoid nerve and vessels Loose connective tissue Infection spreads through lower 3rd molar Tenderness and severe pain in ramus region Trismus and swelling extends from lower border of mandible to zygomatic arch Infection spreads from Pain and trismus Swelling over mandibular 3rd molar, and the temporal region also from maxillary 3rd molar after contaminated Inferior alveolar nerve block No E/O swelling Tenderness dysphagia Oedema in 3rd molar area iii Features - Contd http://dentalebooks.com Infection of the Orofacial Region 131 Contd Spaces Sub-masseteric space iv Surgical treatment Extra oral is mostly used in (Incision and drainage) case of trismus in skin behind angle of mandible Intraoral vertical incision is placed over lower part of anterior border of ramus Pterygomandibular space Temporal space (superfifcial and deep) Extraoral incision is made in skin below angle of mandible Extra oral incision in temporal region, which should be above hair line and 45° to zygomatic arch Intraoral vertical incision is made on anterior and medial aspect of ramus of mandible *Masticatory space infection usually results from- • Infection of the last two molars • External or internal trauma to the mandibular angle region • Non aseptic technique in LA Fig 9.10: Secondary spaces and its boundaries Fig 9.8: Pterygomandibular space and its boundries Fig 9.9: Masticatory space and boundaries Fig 9.11: Site for extra oral incisions to drain abscess http://dentalebooks.com 132 A Concise Textbook of Oral and Maxillofacial Surgery LUDWIG'S ANGINA Features Angina is chocking or suffocating sensation Ludwigs Angina is a firm, acute, toxic cellulitis involving bilaterally the sublingual, submandibular and submental spaces • Always involves all the three - sublingual, submandibular and submental spaces bilaterally • Rapidly developing board-like, brawny, firm, indurated, diffused, non-fluctuating swelling in the floor of the mouth which shows no pitting on pressure (Fig 9.13) • Discomfort and pain while eating, swallowing, breathing, speaking and chewing • Characteristic 'OPEN MOUTH' Appearance with elevated, protruded tongue and elevated floor of mouth • Toxic signs of high fever, rapid pulse, chills, rigor, excessive salivation, trismus and angina • As infection continues, edema of tongue increases and there is high risk of death due to suffocation Etiology • Infections of mandibular molars • Postextraction complication • Compound mandibular fractures • Submandibular gland sialadenitis • Oral soft tissue lacerations Microbiology • Streptococcal infections or mixed flora • Reports also show the presence of Staphylococci, E.Coli, Pseudomonas and certain anaerobes Pathogenesis According to many authors, like Kruger, Topazian and Ludwig, the infection starts in submandibular space and later spreads, but by this it is difficult to explain the pathogenesis Hence, Laskin tells that the infection starts at the sublingual space and spreads bilaterally extending posteriorly over mylohyoid muscle to involve the submandibular and submental spaces at a later stage Further more, the infection spreads to the pharyngeal spaces and the mediastenum (Fig 9.12) Fig 9.13: Clinical view of a patient suffering from Ludwig’s angina (For color version see plate 2) Treatment Fig 9.12: Spread of infection in Ludwig's angina General management of infection is manifested to destroy or inhibit bacterial growth and to increase the physiological defense mechanism of patient – Bed rest is advised, along with hydration – Empirical antibiotics should be administered http://dentalebooks.com Infection of the Orofacial Region – Airway potency and vital sign management (may need tracheostomyLaryngotomy and Cricothyroidotomy/ Tracteotomy are more preferred) – Heat and cold therapy (apply moist heat over the area for 20-30 min/hour) Surgical intervention is instituted for releasing tissue tension and for pus drainage Incisions are made separately for submandibular, sublingual and submental spaces as explained earlier and the pus is collected which is inturn send for gram stain, culture and antibiotic sensitivity test An artery forcep or Hilton's forcep is thrust through the incision and a drain is fixed to enable complete clearing of pus (Hilton's method of Abscess drainage)sublingual and submental space can be approached through the incision line made for the submandibular space, but this is not preferred, as here excision of submandibular muscle has to be done 133 • Fever, nausea, vomiting, chills, rigor, stiffness and constitutional symptoms • Tender and painful eye, paralysis of the extraocular muscles (ophthalmoplegia), edema of eyelid, proptosis, and conjunctivitis Investigations Mainly diagnosed by the clinical features along with skull radiograph, C.T Scan, M.R.I and CSF examination Treatment • Immediate empirical antibiotic therapy like I.V- chloramphenicol, Aminoglycosides, Trimethoprim, Sulphamethazole • In case of abscess surgical drainage via craniotomy procedure is done • Emergency neurosurgical intervention is required Prognosis Prognosis If not treated early and efficiently can lead to death due to aspiration causing severe sepsis COMPLICATIONS OF ODONTOGENIC INFECTION Cavernous Sinus Thrombosis It is a septic thrombosis of cavernous sinus caused due to an infection in the orofacial region, like sinusitis, abscess or cellulitis of the orbit, upper lip, nose, maxilla or dental tissues The classical dangerous area of the face (Triangular area having its base as the upper lip and its apex as the root of nose) has valveless venous drainage which empty itself into the cavernous sinus thus making it easier for any infection of the region to enter into the sinus Features • Suddenly occurring, widely spreading and severe in nature Invariably fatal whereby death occurs due to brain abscess or meningitis Brain Abscess It is the second most common neurological complication, after meningitis of head and neck infections, occurring from bacterimia accompanying odontogenic infections Features • Inflammation, localized edema with septic thrombosis and abscess may develop • Headache, nausea, vomiting, chills, rigor, fever • Sometimes headache is the only symptoms present • Papilledema • Hemiplegia and hemianopsia • Convulsion and abberant nerve palsy • Stupor, confusion and subtle change in personality is also seen http://dentalebooks.com 134 A Concise Textbook of Oral and Maxillofacial Surgery Treatment • Antibiotic therapy (I.V - chloramphenicol) along with steroid ( Dexamethazone and mannitol) • Lumbar puncture if meningitis is suspected • Surgical drainage of abscess by craniotomy Meningitis It is the most common neurologic complications in orofacial infections whereby bacteria infect the arachnoid mater, pia mater and CSF Features • Headache, fever, nausea, vomiting, chill, rigor • Pain and stiffness of neck and back • Confusion, stupor, seizure and coma • Kernig's sign positive (strong resistance is felt when an attempt is made to extend the knee from flexed thigh position) • Brudzinski's sign positive (abrupt neck flexion in supine patient resulting in involuntary flexion of knee Treatment • Lumbar puncture is done and CSF is collected for examination • Antibiotic therapy is preferred to surgical intervention Predisposing factors of the condition • Improper oral hygiene • Occlusal trauma • Gingival infection • Food lodgment • Reduced body resistance Types of Pericoronitis Pericoronitis can be classified into types depending upon the features present: i Acute: Here all the classical features are present ii Subacute: Here the classical symptoms have subsided but certain signs are still present along with the presence of a sinus tract iii Chronic: Here most of the features have subsided but a distinct fistulous tract is present Features • Crypt like area is formed between the tooth and operculum which favours food and debris lodgement and thus microbial proliferation (Fig 9.14) • Severe localized or radiating pain • Distinct extraoral and/or intraoral swelling near the angle of mandible of the affected side and/or opposite to 3rd molar respectively • Submandibular lymphadenopathy and lymphadenitis Mediastenitis It is an extension of infection from deep neck spaces into the mediastenum, which is caused as a very late complication thereby causing chest pain, dyspnea, unremitting fever and characteristic mediastenal widening in radiograph The condition is treated by long term antibiotic therapy and surgical drainage of mediastenum PERICORONITIS It is a infection of the operculum covering the partially erupted permanent teeth specially the mandibular 3rd molar Fig 9.14: Clinical view of pericoronitis (For color version see plate 2) http://dentalebooks.com Infection of the Orofacial Region 135 Fig 9.16: Pericoronitis Fig 9.15: Radiographic view of pericoronitis (For color version see plate 2) • Fever, malaise, increased pulse rate, increased respiratory rate, dysphagia • Sloughing and ulceration of operculum, halitosis and trismus • Fistulous tract which may or may not be draining may be seen Treatment • In case of Acute Pericoronitis, general management of an odontogenic infection complete bed rest, soft nutritious diet and proper oral hygiene with use of mouth rinses, is advised • Proper use of antibiotic and analgesic should be instituted - Penicillin, Doxycycline, and Metrinidazole is preferred • Thorough debridement of tissues with chlorhexidine irrigation and hot saline mouth wash • In certain cases, surgical excision of the operculum - operculectomy (Fig 9.17) or use of caustic agents or electrocautery with or without extraction of the offending, opposing maxillary tooth is adivised • In case of chronic Pericoronitis, proper antibiotic and analgesic with or without extraction of offending, opposing maxillary tooth is recommended Fig 9.17: Operculectomy Fig 9.18: Postoperative PERITONSILLAR ABSCESS (QUINSY) It is a localized infection of the tissues between the tonsils and superior constrictor muscles and between the anterior and posterior pillars of fauces http://dentalebooks.com 136 A Concise Textbook of Oral and Maxillofacial Surgery Features FURTHER READING • Infection spreads from an acute pericoronal abscess which is present near the tonsils or from an infection near the supratonsillar fossa • Pain near the throat and ear of the affected side • Fever, dysphagia, trismus, drooping of saliva, altered speech, and difficulty in swallowing Treatment General management of an infection with soft diet and proper antibiotics and analgesics are recommended Surgical drainage by an incision into the most prominent part of the soft palate to drain the abscess If not treated properly severe oedema affecting the tongue and epiglottis may occur which may even lead to death Shafer-Hine-Levy—Shafer’s textbook of oral pathology, 5th ed 2006 Killey HC, Seword GR, Kay LN— An outline of oral surgery, part I Topozian RG, Goldberg MG—oral and maxillofacial infection, 3rd ed 1999 Peterson, Ellis Hupp, Tucker—Contemporary oral and Maxilllofacial Surgery, 4th ed, 2006 Danier M Laskin—Oral and Maxillofacial surgery The biomedical and clinical basis for surgical practice Vol Archer WH—Oral and maxillofacial surgery, 5th ed vol http://dentalebooks.com ... Deepika Kenkere MDS, Prof and Head-Department of Oral and Maxillofacial Surgery, Sri Rajiv Gandhi College of Dental Sciences and Hospital, Bangalore and Dr N Srinath MDS, Department of Oral and Maxillofacial. .. 16 5 13 Maxillary Sinus and Its Disorders 17 1 14 Cysts of the Oral Cavity 17 9 15 Tumors of the Oral Cavity and Oral Malignancies 18 6 16 Salivary Gland and Its Disorders 19 3 17 Temporomandibular... Rajarshi Banerjee BDS, MDS, MOMS, RCPS Oral and Maxillofacial Surgeon Prof and Head-Department of Oral and Maxillofacial Surgery Dr BR Ambedkar Institute of Dental Sciences and Hospital, Patna